Hepatocytes contribute to residual glucose production in a mouse model for glycogen storage disease type Ia
G6PC mRNA Therapy Positively Regulates Fasting Blood Glucose and Decreases Liver Abnormalities in a Mouse Model of Glycogen Storage Disease 1a - ScienceDirect
Lessons from new mouse models of glycogen storage disease type 1a in relation to the time course and organ specificity of the disease. - Abstract - Europe PMC
Analyzing the Distribution of Mutations for Glycogen Storage Disease Type 1a in Turkey and Suggested Gene Therapy Methods for It
G6PC indicated poor prognosis in cervical cancer and promoted cervical carcinogenesis in vitro and in vivo | Reproductive Biology and Endocrinology | Full Text
Mouse G6pc activation kit by CRISPRa – GA201504 | OriGene